How can a diabetic lose belly fat

How can a diabetic lose belly fat

Diabetes: Why Should You Lose Weight At The Waist?

Susceptibility to diabetes is associated not only with being overweight but also with the location of adipose tissue in the body.

Diabetes and obesity go hand in hand, and the number of people affected by both of these problems continues to grow. It is estimated that 2/3 of the US population is overweight or obese. A similar trend is visible in Europe. At the beginning of the century, 171 million people worldwide had adult diabetes. Scientists estimate that by 2030 this number will reach 360 million. Understanding the mechanisms of the disease, its relationship to overweight and insulin resistance, will allow the development of more effective methods of diabetes prevention.


Diabetes mellitus is described as a disease involving a reduced production of insulin in the pancreas and the tissue resistance to it. This does not mean that excess body fat directly translates into hyperglycemia. The pancreatic islets are able to produce enough insulin to effectively regulate blood glucose levels, even with impaired tissue response to insulin. Diabetes mellitus develops when islet cells are unable to compensate for the sensitivity of the tissues with increased insulin production. Insulin resistance leads to elevated levels of fatty acids in the blood, decreased transport of glucose to the muscles, and impaired breakdown of fat, which in turn causes excessive production of glucose in the liver. Every obese person has some degree of insulin resistance.

Reduced tissue sensitivity to insulin occurs naturally during puberty, pregnancy, and as the body ages. During our life, our lifestyle, physical activity, and the amount of sugar consumed also change. One of the main factors changing our metabolism is overweight.

Insulin resistance is a function of BMI and increases with increasing BMI. BMI (Body Mass Index) is a factor that determines body mass in relation to height. It is calculated by dividing your weight in kilograms by your height in meters squared. Values between 18.5 and 24.9 are considered normal, 25–29.9 are overweight, over 30 are obese, and over 40 are severely obese. The higher the BMI, the greater the health consequences.


Susceptibility to diabetes also depends on the location of adipose tissue in the body. People with an apple shape, where fat accumulates around the abdomen and chest, have higher insulin resistance than those with fat on the periphery of the body in the subcutaneous tissue.

Belly fat is more resistant to insulin and is therefore more important in the mechanism of insulin resistance.


The pathophysiological mechanism of type 2 diabetes remains debatable, but it is clear that with increasing amounts of fat stored, tissue sensitivity to insulin decreases.

Many people predetermined to develop type 2 diabetes tend to accumulate visceral fat. These people have an impaired ability to accumulate fat in the subcutaneous tissue. An extreme example illustrating this mechanism is lipodystrophy. People affected by this disease cannot accumulate fat in the subcutaneous tissue and any weight loss causes fat to accumulate in the abdominal cavity and in organs where it should not be, e.g. the liver. These individuals show a significant degree of tissue resistance to insulin.

On the other extreme, there are people, especially women, who, despite BMI proving obesity, have normal blood lipids and sugar levels, and normal tissue sensitivity to insulin. These people are distinguished by a different distribution of adipose tissue, i.e. the dominance of subcutaneous fat accumulation.


Studies conducted in the USA and Canada have shown that the prevalence of diabetes clearly correlates with the degree and type of obesity. A group of 15,532 women belonging to TOPS Club groups - a charity organization dedicated to helping to get rid of excess weight - was gathered. Severe obesity increased the likelihood of diabetes more than threefold. To a similar degree, the type of obesity was associated with diabetes. People with an apple structure, i.e. adipose tissue located around the abdomen and chest, suffered from diabetes three times more often than those with the pear type, i.e. the dominant peripheral adipose tissue. Comparing both body types, 62 percent of women with central obesity had a diabetic glucose tolerance curve, while laboratory tests showed no suspicion of diabetes among people with peripheral obesity.

There are also differences between the sexes. Men create diabetes at a lower BMI than ladies. This is also due to the greater potential for fat accumulation in the subcutaneous tissue in women.


In finding confirmation of the direct impact of obesity on the onset of type 2 diabetes, the question arises as to whether weight loss through surgical intervention or through diet can reverse diabetes. The answer is yes. Studies show that a loss of about 38 kg in a person with a BMI of about 48 kg / m 2 leads to diabetes remission in three out of four patients. In short, the greater the weight loss, and the shorter the duration of diabetes, the greater the chance of diabetes remission.

The diet itself also turns out to be effective. In the Newcastle group, 11 patients showed normalization of glucose levels after a week of a 600 kcal/day diet. Hepatic triglycerides dropped by 30 percent. Although the weight loss was only 4 percent. The liver's response to insulin improved on average from 43 to 74 percent. It is clear that such a restrictive diet is acceptable for a short time, but the results of this study, while requiring confirmation, seem promising.

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